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ritastrakosha

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About ritastrakosha

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  1. They are psychopaths for sure. Evil is psychopathic and antisocial. But being antisocial or psychopathic does not necessarily mean that you are a liar. I know of quite sincere psychopaths. On the other hand, people with personality disorders that predispose to schizophrenia (schizoid personality disorder for example) are often not able to distinguish between reality and their imagination, or wishes, so they lie. An evil liar would have a mix of personality disorders. Peope who call themselves evil liars, may do so as a joke, but there is a saying "There is a grain of truth in every joke". Do you know evil liars around you? If yes, do they have diagnosed personality disorders?
  2. So far in literature homosexuality has been mostly related to either gender nonconformity (by Michael Bailey especially ) or high sexual drive (by religious therapists mainly). But there are cases of homosexuality where the person has neither. The following is one such case: "At the age of 8 or 9, and long before distinct sexual feelings declared themselves, I felt a friendly attraction toward my own sex, and this developed after the age of puberty into a passionate sense of love. I was a day-boarder at school and heard little of school-talk on sex-subjects, was very reserved and modest besides; no elder person or parent ever spoke to me on such matters; and the passion for my own sex developed gradually, utterly uninfluenced from the outside. I never even, during this period, and a good deal later, learned the practice of masturbation. My own sexual nature was a mystery to me. I found myself cut off from the understanding of others, felt myself an outcast, and, with a highly loving and clingy temperament, was intensely miserable… As a boy I was attracted in general rather by boys older than myself; after leaving school I still fell in love, in a romantic vain, with comrades of my own standing… My chief desire in love is bodily nearness or contact…." Same-sex sexual attractions in this case may emerge from an interaction between sex drive development and social development during early adolescence, through associative learning mechanisms. The affiliation hypothesis of homoerotic motivation in humans asserts that social bonding is one of the main drivers of homosexual behavior. Fleischman et al. have argued that in women and men, progesterone, a hormone shown to promote affiliative bonding, is positively associated with homoerotic motivation. Oxytocin, another hormone related to bonding, induced homosexual preference in male rats. Homosexual men in comparison to heterosexual ones display higher sensitivity to oxytocin's enhancing impact on social approach tendencies. “Affiliative” homosexuality may be the result of a phenomenon known as synesthesia, in which stimulation of one sensory or cognitive pathway leads to automatic, involuntary experiences in a second sensory or cognitive pathway. There are at least 60 different types of synesthesia. It may affect up to 4.4% of the population. In a survey with transgender persons Jay Pierce found that 42% of transgender participants endorsed synesthesia compared with 16% of heterosexual participants. He proposes that synesthesia and transgender identity may share a common biological cause. Cytowic has noted that by his estimate at least ten percent of synesthetes are gay or lesbian (while the prevalence of gay or lesbian identity in the population is around 1-2 %) . There are several models that explain synesthesia. The associative learning model asserts that the inducer (peer attachment in our case) and concurrent stimuli (spontaneous sexual arousal) have been present in a correlated, or “contingent,” fashion in the synaesthete's environment; hence presentation of the inducer alone now activates the representation of the concurrent. Adolescents experience both increasing peer attachment and spontaneous sexual arousals. This could lead to a synesthesia of peer attachment → sexual arousal. Synesthesia may take years to fully develop. While the high sexual drive→homosexual attraction pathway may be driven by the dopamine system, the strong peer attachment→homosexual attraction pathway may be driven by the serotonin system. These two pathways to homosexual attraction may correspond to two different personality types: extroverts may be more prone to high sexual drive→ homosexual attractions pathway while introverts may be more prone to strong peer attachment→homosexual attractions pathway. Dopamine enhances appetitive conditioning, while serotonin is known for enhancing aversive conditioning. Affiliative homosexual attractions may be the result of aversive conditioning: the spontaneous sexual arousals get weaker in the presence of opposite sex individuals due to high anxiety (synesthetes have higher rates of anxiety disorders than non-synesthetes), but remain in the presence of same-sex peers, and as a result a contingency arises between sexual arousal and same-sex peers. Strong peer attachment and gender conformity in this case bring the opposite effect of what is expected from the “exotic becomes erotic” model of Bem. Introverted personalities with high anxiety and strong gender conformity, peer attachment may perceive the exotic as threatening, aversive. The cross-activation model of synesthesia proposes that excess connectivity between functional areas of the cortex allows activation in one cortical area to directly trigger activation in another. The disinhibited feedback model proposes that synesthetic sensations are caused by disinhibited feedback from higher cortical areas failing to suppress non-relevant activation from lower cortical areas. The serotonin hypothesis of synesthesia proposes that the presence of excessive levels of serotonin increases the excitability and connectedness of sensory brain regions and leads to synesthesia. The immune hypothesis of synesthesia posits that the immune system could have a direct influence on the excessive cross-activation and disinhibited excitatory neuronal activity leading to synesthesia. It hypothesizes that the immune system could be involved in the regulation of brain development leading to synesthesia, specifically through its systems: complement system, cytokines (proteins released by different immune cells including mast cells), major histocompatibility complex (which are also involved in inflammatory processes).
  3. Can you support the statement "you are a moral crusader" with facts? So as we are scientific here :-).
  4. Michael Bailey, a renowned sexologist, has recently written an opinion article in the Archives of Sexual Behavior, titled "How to ruin sex research". He writes that sex research is being hampered by ideology and scientists should not bow to ideology. He urges for the controversial ideas in sex research not to be censored. Quote: If people knew which ideas had merit and which did not even before the ideas had been thoroughly discussed and tested, then we would not need science. What do you think?
  5. Could this also happen to humans? Sci Rep. 2019 Mar 18;9(1):4837. doi: 10.1038/s41598-019-41258-2. Prenatal Allergen Exposure Perturbs Sexual Differentiation and Programs Lifelong Changes in Adult Social and Sexual Behavior. Lenz KM1,2,3, Pickett LA4,5, Wright CL4,5, Galan A6, McCarthy MM4,5. Author information Abstract Sexual differentiation is the early life process by which the brain is prepared for male or female typical behaviors, and is directed by sex chromosomes, hormones and early life experiences. We have recently found that innate immune cells residing in the brain, including microglia and mast cells, are more numerous in the male than female rat brain. Neuroimmune cells are also key participants in the sexual differentiation process, specifically organizing the synaptic development of the preoptic area and leading to male-typical sexualbehavior in adulthood. Mast cells are known for their roles in allergic responses, thus in this study we sought to determine if exposure to an allergic response of the pregnant female in utero would alter the sexual differentiation of the preoptic area of offspring and resulting sociosexual behavior in later life. Pregnant rats were sensitized to ovalbumin (OVA), bred, and challenged intranasally with OVA on gestational day 15, which produced robust allergic inflammation, as measured by elevated immunoglobulin E. Offspring of these challenged mother rats were assessed relative to control rats in the early neonatal period for mast cell and microglia activation within their brains, downstream dendritic spine patterning on POA neurons, or grown to adulthood to assess behavior and dendritic spines. In utero exposure to allergic inflammation increased mast cell and microglia activation in the neonatal brain, and led to masculinization of dendritic spine density in the female POA. In adulthood, OVA-exposed females showed an increase in male-typical mounting behavior relative to control females. In contrast, OVA-exposed males showed evidence of dysmasculinization, including reduced microglia activation, reduced neonatal dendritic spine density, decreased male-typical copulatory behavior, and decreased olfactory preference for female-typical cues. Together these studies show that early life allergic events may contribute to natural variations in both male and female sexual behavior, potentially via underlying effects on brain-resident mast cells.
  6. I answered you on that. Vaccinations have decreased epidemics but not to the extent that is claimed by the pharmaceutical industry. The decrease in infectious diseases began before vaccine use became widespread.
  7. I don`t understand you. What ideology? One way paracetamol is thought to contribute to autism is by depleting glutathione, which detoxifies (aluminum among others). Paracetamol is given after vaccination.
  8. So you say that a population where let's say 10% of the pairs are homosexual and 90% of the pairs are heterosexual the family is stronger and children better cared for than in a population where 100% of the pairs are heterosexual. Homosexual pairs are not better equipped than heterosexual pairs to raise children. It would be evolutionary better for children to be raised by male-female pairs instead of male-male or female-female pairs. Millions of years of evolution have led to female-male pairs being the norm.
  9. It is under review. Yes, there is plenty of homosexual behavior, like there is plenty of different kinds of behavior. So, what? 2000 years of brainwashing seems a long time. I know about causation and correlation. The study has addressed this by explaining at the physiological level how stress leads to homosexuality. I am an atheist.
  10. Another interesting thing is, why are you so obsessed with psychology and psychiatry to be a member of this forum? Keep focused on the question. The answers to your dilemmas are in the study.
  11. I mean the injection route is 1 million times more efficient at increasing aluminum content in immune cells. What does the title you cite make clear? Artificial light is a stress source. There is abundant research on this. There are multiple other stress sources linked to homosexuality, whom the journalist does not mention, perhaps because of feeling uncomfortable around them, like drug use for example?
  12. My question is: What do the members of this forum think about the hypothesis that diet and stress cause homosexuality? If they wish to know why I think so, they have all the information in the shared link. The link is to a non-profit science archive. I have to link because the study has over 60 pages of interlinked information and arguments. It has new research and ideas compared to the one I shared last year. There are new studies published all the time on homosexuality. Many scientists see it as researchable, hence discussable.
  13. I was watching an interview with Chris Exley yesterday, who has studied aluminum for 35 years. He said that they haven't found in any other body parts such high levels of aluminum as in the glia of the brains of autistic children. Exceley has also done studies on aluminum and Alzheimer and says that the link is strong. He is not funded by the industry, the states or the charities because there is no money in such study, on the contrary. Meanwhile his study has been downloaded 250,000 times. Vaccines contribute to the decrease of epidemics, but their contribution is smaller than thought (much smaller according to some). In an international science conference on vaccines held in Portugal this summer one of the scientists (Romain Gherardi?) was saying that compared to the injection route one would have to ingest 1 million times more aluminum to have the same accumulation of it in the immune cells.
  14. I have had measles myself. Almost all children got measles at my time. It was like having a severe influenza. It lasted for several days and it was over. Measles causes severe complications in 1 in 2000 children. Taking vitamin A can halve the complications from measles. Today, children with allergies, asthma have to suffer all their life. The decline in infectious diseases began before the mass use of vaccines and was rather due to the improved nutrition and sanitation. You may be interested to read the below papers on aluminum. Around 40% of children in developed countries have an immune disorder today (food allergies, asthma, eczema, autism etc. which are all immune related disorders). Why all this sickness? J Inorg Biochem. 2011 Nov;105(11):1489-99. doi: 10.1016/j.jinorgbio.2011.08.008. Epub 2011 Aug 23. Do aluminum vaccine adjuvants contribute to the rising prevalence of autism? Tomljenovic L1, Shaw CA. Abstract Autism spectrum disorders (ASD) are serious multisystem developmental disorders and an urgent global public health concern. Dysfunctional immunity and impaired brain function are core deficits in ASD. Aluminum (Al), the most commonly used vaccine adjuvant, is a demonstrated neurotoxin and a strong immune stimulator. Hence, adjuvant Al has the potential to induce neuroimmune disorders. When assessing adjuvant toxicity in children, two key points ought to be considered: (i) children should not be viewed as "small adults" as their unique physiology makes them much more vulnerable to toxic insults; and (ii) if exposure to Al from only few vaccines can lead to cognitive impairment and autoimmunity in adults, is it unreasonable to question whether the current pediatric schedules, often containing 18 Al adjuvanted vaccines, are safe for children? By applying Hill's criteria for establishing causality between exposure and outcome we investigated whether exposure to Al from vaccines could be contributing to the rise in ASD prevalence in the Western world. Our results show that: (i) children from countries with the highest ASD prevalence appear to have the highest exposure to Al from vaccines; (ii) the increase in exposure to Al adjuvants significantly correlates with the increase in ASD prevalence in the United States observed over the last two decades (Pearson r=0.92, p<0.0001); and (iii) a significant correlation exists between the amounts of Al administered to preschool children and the current prevalence of ASD in seven Western countries, particularly at 3-4 months of age (Pearson r=0.89-0.94, p=0.0018-0.0248). The application of the Hill's criteria to these data indicates that the correlation between Al in vaccines and ASD may be causal. Because children represent a fraction of the population most at risk for complications following exposure to Al, a more rigorous evaluation of Al adjuvant safety seems warranted. Aluminium in brain tissue in autism Author links open overlay panelMatthewMoldaDorcasUmarbAndrewKingcChristopherExleya Show more https://doi.org/10.1016/j.jtemb.2017.11.012Get rights and content Under a Creative Commons license open access Abstract Autism spectrum disorder is a neurodevelopmental disorder of unknown aetiology. It is suggested to involve both genetic susceptibility and environmental factors including in the latter environmental toxins. Human exposure to the environmental toxin aluminium has been linked, if tentatively, to autism spectrum disorder. Herein we have used transversely heated graphite furnace atomic absorption spectrometry to measure, for the first time, the aluminium content of brain tissue from donors with a diagnosis of autism. We have also used an aluminium-selective fluor to identify aluminium in brain tissue using fluorescence microscopy. The aluminium content of brain tissue in autism was consistently high. The mean (standard deviation) aluminium content across all 5 individuals for each lobe were 3.82(5.42), 2.30(2.00), 2.79(4.05) and 3.82(5.17) μg/g dry wt. for the occipital, frontal, temporal and parietal lobes respectively. These are some of the highest values for aluminium in human brain tissue yet recorded and one has to question why, for example, the aluminium content of the occipital lobe of a 15 year old boy would be 8.74 (11.59) μg/g dry wt.? Aluminium-selective fluorescence microscopy was used to identify aluminium in brain tissue in 10 donors. While aluminium was imaged associated with neurones it appeared to be present intracellularly in microglia-like cells and other inflammatory non-neuronal cells in the meninges, vasculature, grey and white matter. The pre-eminence of intracellular aluminium associated with non-neuronal cells was a standout observation in autism brain tissue and may offer clues as to both the origin of the brain aluminium as well as a putative role in autism spectrum disorder. Biopersistence and Brain Translocation of Aluminum Adjuvants of Vaccines Romain Kroum Gherardi,1,* Housam Eidi,1 Guillemette Crépeaux,1 François Jerome Authier,1 andJosette Cadusseau1 Author information Article notes Copyright and License information Disclaimer Abstract Aluminum oxyhydroxide (alum) is a crystalline compound widely used as an immunological adjuvant of vaccines. Concerns linked to the use of alum particles emerged following recognition of their causative role in the so-called macrophagic myofasciitis (MMF) lesion detected in patients with myalgic encephalomyelitis/chronic fatigue/syndrome. MMF revealed an unexpectedly long-lasting biopersistence of alum within immune cells in presumably susceptible individuals, stressing the previous fundamental misconception of its biodisposition. We previously showed that poorly biodegradable aluminum-coated particles injected into muscle are promptly phagocytosed in muscle and the draining lymph nodes, and can disseminate within phagocytic cells throughout the body and slowly accumulate in brain. This strongly suggests that long-term adjuvant biopersistence within phagocytic cells is a prerequisite for slow brain translocation and delayed neurotoxicity. The understanding of basic mechanisms of particle biopersistence and brain translocation represents a major health challenge, since it could help to define susceptibility factors to develop chronic neurotoxic damage. Biopersistence of alum may be linked to its lysosome-destabilizing effect, which is likely due to direct crystal-induced rupture of phagolysosomal membranes. Macrophages that continuously perceive foreign particles in their cytosol will likely reiterate, with variable interindividual efficiency, a dedicated form of autophagy (xenophagy) until they dispose of alien materials. Successful compartmentalization of particles within double membrane autophagosomes and subsequent fusion with repaired and re-acidified lysosomes will expose alum to lysosomal acidic pH, the sole factor that can solubilize alum particles. Brain translocation of alum particles is linked to a Trojan horse mechanism previously described for infectious particles (HIV, HCV), that obeys to CCL2, signaling the major inflammatory monocyte chemoattractant.
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