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rakuenso

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Posts posted by rakuenso

  1. Also note that a species crossing flu doesn't have to infect humans. As an H9 variant can also mutate and infect a bird with the H5 strain as opposed to the commonly accepted H5 affecting H9 human

  2. The H5N1 strain obviously is not very effective at human-human transmissions, else it would've spread all over the world by now. I thought he meant the current not so deadly H9 strains, which can easily be transferred from person to person.

  3. Wow if we knew what decided death we would've found a cure already. Obviously that isn't the case. Take the example of viruses, are they dead or alive, nobody knows. For multicellular organisms, a dead organism is an organism that no longer affects the population.

  4. Another Bush answer would be to nuke india,

     

    however, there has to be delicate balance between vaccine production.

     

    We can't both stock up vaccine for the H5N1 strain without straining the production capabilities of the "normal" strain. You can see how this would pose a problem, as if we augment falsely the production of vaccines for the H5N1 strain, we won't be able to stock up on the normal strain. Where millions more might die as a result of lacking the normal vaccine. Secondly, we can't just make a new vaccine for instance the day we find that there's a pandemic, these vaccines take months to prepare.

  5. yes can increased definition could mean more production of a certain enzyme. But its not the only possibility. It could also be that bifenthrin doesn't inhibit the gene expression, but rather inhibits the body's ability to use that enzyme. A typical sign of increased gene expression could and likely will result in an increase in the enzyme levels. You can find out by using a microarray for multiple genes or a variant of PCR for a single gene (The procedure is far too complicated for me understand, anyone else mind clarifying?)

  6. So you are you talking about cancerous adrenal cells eh?

     

    Normally it works like this... Gene expression is a tightly regulated mechanism that can go be maintained by many pathways. I.E. JaK-STAT, however in tumour cells many things can happen

     

    either genes responsible for inhibiting mitosis are deactivated/defective

    or pro-mitotic genes are activated/dysregulated

    there are also apoptic genes that can be disturbed, for example leading to the failure of Fas and FasL signaling.

    and many other possibilities

     

    Gene expression is controlled by a set of general transcription factors/proteins that mediate which corresponding genes are turned on/off. The Basal Core promoter consists of a TATA box that TBP binds to. The upstream promoter can be as far away as 200bp. While enhancers can reach tens of thousands bps upstream (maybe downstream even, anyone?). Anyways, a gene can only be expressed, thats is made into a primary mRNA transcript if the GTFs are bound to the promoter (in almost all cases, I haven't heard of any exceptions). Once the GTF is bound, the polymerases come in and do their job.

     

    Wikipedia can probably explain it better than I, since its such a broad topic.

  7. college and university are different levels? I say we start it by finding the integral of |x|^-|x| from -infinity to +infinity :D

     

    even though i'm pretty sure there's no algebraic way to solve it

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