Joshua201

If you don't work there, it's going to be difficult obtaining permission to use their patient data. You could find out what applies in your country and what the appropriate method is

An increase in the hydrostatic pressure over that opposed by intravascular oncotic pressure would normally cause fluid loss from the vessels

Summarising the ppt material on paper helps a great deal too. Get the idea, teach someone, it'll stick better that way. Studied Obstetrics abs Gynaecology that way myself

There's really no correlation between cranial size and IQ. Also a large cranial cavity doesn't necessarily translate to a larger brain.

Not so much. Need help with something?

Same way temperature homeostasis is accomplished

where can i find one?

in your school

Diarrhoea and Septic Shock occuring together shows that the diarrhoea is of an infective origin. Same mechanism responsible for the shock 'll be responsible for the diarrhoea

Basically it is assumed that the saliva of the mosquitos triggers a weak allergic reaction. However, it should be noted that there are apparently histamine-dependend as well as histamine-independent pathways resulting in the itch. The latter mechanisms is not quite clear, but seems to play a major role.

true, histamines are responsible

Can i just buy the e-book ? I need links & the names for tge books for 2nd year General medicine.

looking to buy books, amazon 'll be the site of choice

9/10 if you ask me. You've to think like a scientist to be one.

I would go and consult an ophthamologist.

That is what I would also recommend

same recommendations

From Wiki.

The United States Food and Drug Administration,[2] World Health Organization,[3] International College of Nutrition,[4] the United States Department of Health and Human Services,[5] American Dietetic Association,[6] American Heart Association,[7] British National Health Service,[8] and Dietitians of Canada[6] recommend against the consumption of significant amounts of coconut oil due to its high levels of saturated fat.

 

(Which is a pity, because I really like coconut)

one strikes out the other

In all honesty, if I have to do a quick kick due to saturated fats as opposed to alzheimers; get me out of here! At least you get the message.

think they're just proteins or other particles floating in the vitreous or aqueous humors.remember they're recycled hence are connected to the body's general circulation, so

stuff

might get in

Don't know that it affects seminal production, but it will certainly increase the amount of leaping about screaming bloody murder you do in a day. Try to wash your hands before smacking yourself in the forehead since you don't want it in your eyes either.

totally agree. no effect on that

I think the answer is no, but I have some wax for my braces which I left exposed to the air in the bathroom. Then I cleaned part of my trumpet out, and left a rusty metal snake on top of the wax. Anyways, I rinsed off the wax under hot water, and I just want to double check that the rust can't hurt me when I use it in my mouth.

unless you ingest broken off pieces or have an area of eroded epithelium in your mouth.

Hi:

 

There are many cases of patient who are "clinically dead" and are then brought back to life. During this 'death', these patients are able to perceive the universe in locations beyond the range of normal perception. For example, such a patient can see what is happening in other parts of the hospital and outside the hospital. What the patient perceives is often confirmed by real events. In addition, patients who are born blind can see during this out-of-body-experience and patients born deaf can hear.

 

What are the scientific explanations for the above interesting facts?

 

How can one perceive beyond normal sensory mechanisms?

 

How can one born without sight be able to see during an OBE?

 

How can one born without hearing be able to hear during an OBE?

 

After reading about these cases, I'm no longer convinced there is such a thing as 'death'. No matter how depressed I am, I'll never jump off a cliff because, the resulting OBE maybe a lot more painful then any agony I could experience while 'alive'.

 

 

Thanks,

 

GX

you could as well do research to find out what the blind and deaf dream about on a normal day.

and as for your conviction, ask the cadavers in med school

There's something of a war of words going on here. Technically speaking, the cell death is amount and duration dependent, and not due to the alcohol directly, in either case.

 

As it's a CNS depressant, with enough of it during one night, assuming you don't die, you could easily be left with permanent brain damage (through cell death), due to loss of circulation/lack of breathing. With enough consumed over a long enough duration, brain cell death may also occur, via a number of paths (lack of thiamine, high concentrations of circulating ammonia, etc.). In both cases, it's due to things downstream of the alcohol consumption, rather than the alcohol directly, but you could make an argument either way as to whether alcohol "caused" the cell death or not.

good point

Only the weak ones

seriously ??

I did a short research paper on this topic for one of my undergrad courses, specifically on how alcohol affects the NMDA receptor. Long story short, GABAergic and NEergic neurons normally regulate glutamate using NMDA receptors. However, alcohol blocks NMDA receptors causing disinhibition of excitatory pathways, leading to excitotoxicity. When pregnant rats were administered alcohol, their infant rats displayed fetal alcohol syndrome and increased apoptosis. However, the cells damaged from excitotoxicity usually can repair themselves but if not, alternate neural connections are formed. One thing to keep in mind though is age because mature adult brains tend to exhibit greater sensitivity than immature non-adult brains, although this can be attributed to the massive redundancy in immature non-adult brains that have not yet undergone natural widespread apoptosis. I suppose a limitation to this is the concentration of NMDA receptors varies across brain regions and as we know, the brain does not have simply one type of neuron. Overall, there is an observable excitotoxic effect only in certain brain regions with a high concentration of NMDA receptors, such as the hippocampus, which helps to explain the cognitive impairments seen in Korsakoff's Syndrome.

 

Concise pathway model

GABAnergic intended?

For atlases, try Netters and Grant's.

Then for the main texts, Gray's anatomy and Clinically oriented anatomy is the best way to go.

Other online resources are particularly helpful as well.

totally agree

The book writer may not be perfect for every detail. I think that your understanding is correct, which is enough.

It depends on the focus of the textbook, I presume. Most distinguish between afferent and efferent pathways, though.

I think it varies from one book to another

It depends on the author and his area of concentration.He might be an anatomist or a surgeon or perhaps a physiologist. Their methods of description varies. Most differentiate between afferent and efferent pathways as stated above.

Being introduced to the brain science, one typically learns about line detector neurons in area V1. But I don't recall learning about curve detector neurons. Are there any?

all are part of the functions of line receptors. they function by contrast. curve detection is achieved by same neurons.

Physics would be the perfect choice considering your interests. And also its wider giving you more choices for post graduate studies

proteins are needed for transport across a membrane

I also prefer Lehninger. For me is the best biochemistry textbook!

lehninger, voet, and devlin are good choices