"Baric’s study on the SHC014-chimeric coronavirus began before the moratorium was announced, and the NIH allowed it to proceed during a review process, which eventually led to the conclusion that the work did not fall under the new restrictions, Baric told Nature. But some researchers, like Wain-Hobson, disagree with that decision.
The debate comes down to how informative the results are. “The only impact of this work is the creation, in a lab, of a new, non-natural risk,” Richard Ebright, a molecular biologist and biodefence expert at Rutgers University, told Nature.
But I guess Richard Ebright and Simon Wain-Hobson are just conspiracy theorists.The canned response is tired.
The reason the editors note was placed on this paper was because in multiple interviews this paper along with 2 others were presented by Professor Francis Boyle, Professor of International Law at University of Illinois College of Law, author of U.S. implementing legislation for 1972 Biological Weapons Convention, the Biological Weapons Anti-Terrorism Act of 1989 as proving dual use/gain of function bioweapon research. Those are his words not mine. You should read his book Biowarfare and Terrorism and after you are done with that you should read Seymour Hersh's 1968 classic Chemical and Biological Warfare they are illuminating.
You can try this one on for size too https://www.nature.com/news/engineered-bat-virus-stirs-debate-over-risky-research-1.18787
Its easy to name call but maybe instead of throwing out the tired "conspiracy theory" pejorative you should actually step back and think about this with an open scientific mind. I remember when calling a Nobel Prize winner a conspiracy theorist was risky business but I guess Luc Montagnier doesn't get any respect.
Do you even know the origin of the term "conspiracy theory"?
You act as if this GOF research isn't happening. In case you didn't know there was a lot, a lot, a lot of opposition to this type research. Much of it stemming from the H5N1 and H7N9 research of Ron Fouchier was doing using ferrets to create receptor affinity https://www.sciencemag.org/news/2011/11/scientists-brace-media-storm-around-controversial-flu-studies and https://www.nature.com/articles/500150a
Take for example a case from 1977 https://mbio.asm.org/content/6/4/e01013-15 Professor Osterholm confirmed on video that the 1977 outbreak came from a lab release.
I could hand you stacks of studies showing GOF research.
Did you actually read "The proximal origin of SARS-CoV-2" paper. The evidence for zoonotic jump is just not there.
– When a virus manages to infect a new species of host it’s known as a zoonotic jump, a process that generally takes months or even years to complete. The first stage is when a virus infects one individual in a new host species, which is typically a dead-end the first time it happens since there’s no way for the virus to be adapted to a different species’ biology. The second stage of a zoonotic jump is when the virus manages to move from the first new host into more hosts of the new species, which results in some temporary transmission in a localized area – these are known as endemics and generally fizzle out the first few times they happen as the virus adapts to its new host species, and mutations win or lose the survival battle. The final stage, the only time a zoonotic jump is considered complete, is when there’s sustained host-to-host transmission in the new species. These zoonotic jumps have some predictable characteristics, the primary one is that adapting to a new host inevitably requires mutations that weren’t optimal in the old host. And so the virus gets weakened as its initially attempting to jump into a new host species, which is why the above sequence of steps – one new host, a few new hosts that pass it among themselves temporarily, and then finally sustained transmission – takes at least several months if not years to play out, since a good bit of time is required for all three steps to occur. Viral trial-and-error is required for the virus to find the right mutations that will allow it to prosper in a new host species, it’s never been known to just happen magically all at once.
– And so assuming that COVID-19 emerged naturally in a matter of weeks in the middle of a massive urban metropolis the size of New York City, when the host population of bats was hibernating anyways, requires completely ignoring everything we know about how viruses transfer between species. Not only was Wuhan’s population not interacting with bats since they hardly interact with humans in urban situations to begin with, but any possible host bats were sleeping in their caves anyways. And not only would the circumstances of this transfer require rewriting the textbooks on zoonotic jumps if it occurred at all, but beyond that: supposedly not only did a zoonotic jump happen instantly without the necessary steps, but when it hit humans it was extraordinarily virulent from the start, something that’s supposed to take an extensive amount of time to ever happen as mutations go through selective trial-and-error. This trial-and-error takes time and is why viruses have never made a zoonotic jump and been instantly virulent in a new host species like COVID-19 has been in humans. And in 2018, a study looking for past infections by bat coronaviruses in Wuhan found no evidence whatsoever that anyone there had ever been infected by one at all, making the idea that these viruses have been circulating there entirely absurd. Nothing we know about how viruses naturally make zoonotic jumps point to that happening here.