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  1. So we hear that if one consumes too much refined insulin-spiking carbs, it is very likely that a large portion of it will get stored as fat. Simple question. If I eat a large amount of a fatty low glycémic index food, like drinking some full fat cream (30% fat), physiologically, what happens after my body has used what it needs at that time? Does the rest get stored as fat? (I'm it does) but how?
  2. Hello, This topic is becoming a hobby fascination of mine and I've been reading a fair amount of literature on it, which naturally gives rise to some questions. I have recently realised through a simple BG meter I inherited from my T2 diabetes mum, that my BG levels are slightly higher than what is considered to be normal. It seems to be exclusive to when I am fasting. I say this because on numerous occasions I have eaten the barely-imaginable filth that would cause BG's to rocket in just about anyone and tested my BG's within the hour only to find that it is still below 8mmol/l, whereas while fasting, I can get readings ranging between 6-7mmol/l (once it was 7.9 in the early hours of the morning 6-7am). I initially put this down to insulin resistance, or a combination of other metabolic syndromes that I'm not fully aware of. To put things into context, my height is 190cm, I weigh ~90kg (BMI is around 25, but I'm more muscular than average) and I am around 15-18% bodyfat (at a guess). The only sign I show of having any metabolic issues is a persistent layer of subcutaneous fat hanging off my belly that has never seemed to go away, even when I went down to 85kg and was around 12% BF. I run long distances and have completed a marathon and I do cardio every single day in the form of biking, while maintaining gym visits for weight training at least 3 times a week. Now in terms of the fasting, I have sometimes extended these fasts to 3 days at a time, drinking 0 calorie liquids and occasionally topping up my electrolytes and keeping my exercise at the same level as I would while not fasting. While I feel absolutely great by the 3rd day (ketosis, I tell myself, my definitely be highly active by then) I still manage to have BG readings of 6mmol/l. I definitely see changes in my BF visually by the 3rd day, and if I extend it further I would see alot more. My question is, while I do not mind going for long periods without food, is this fasting practice actually doing my pre-diabetic state any favours? It may be important to point out that I am a habitual breakfast skipper too, and I've read various things in the medical literature about this practice inducing some kind of metabolic syndrome, but I don't understand the underlying physiological argument/basis for this. Can anyone also shed any light on this? Sorry I realise my questions aren't totally direct but I was hoping to start off a discussion to help my knowledge along with certain issues related to this topic.
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