garytse86
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This thread is very popular.
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How are signals form insulin integrated with those from adrealine and contraction to regulate the breakdown or synthesis of glycogen in muscle?
I know that adrenaline stimulates the conversion of ATP to cyclic AMP by activating adenyl cyclase, and I think insulin does the opposite. But how are the signals combined before having a net effect? It would be easier if they were both hormones. Is adrenaline a hormone? Because it is a neurotransmitter specifically acting on target tissues (though Wikipedia says it's a hormone as well). Is the integration of signals just a simple "summation" of the effects? But that would mean there is no integration...
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Enzyme kinetics often follow both zero and first order kinetics.
Zero order kinetics are followed when the substrate S has saturated the enzyme.
First order kinetics are followed when the enzyme is not saturated.
Why is this?
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but don't you lose a lot of the accuracy if you do the transformation?
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Hello there. I have just finished a biological experiment on "effect of trypsin concentration on rate of casein hydrolysis"
I have already obtained a graph, and I used a program called "Graphpad Prism" to analyse the data usin nonlinear regression (3rd degree polynomial). I have got all the parameters like sy.x, degrees of freedom... But how do I use these parameters to analyse whether my results are reliable, and by varying enzyme concentration results in an increase in rate?
I have also considered Spearman's rank coefficient but this is only for linear relationships. What else can I do to determine whether my results show regression?
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We have no resonsibility for the past? What do you think about this topic?
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We owe all our prosperity to science, what do you think about this topic?
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Neuromuscular Junction
in Anatomy, Physiology and Neuroscience
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What are the consequences of having an electrical neuromuscular junction, there must be reasons why the nmj is chemical?