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Jeju

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Posts posted by Jeju

  1. With this hypothesis of oxidative phosphorylation, the study for me has been too theorytical rather than being experimental. I seems to agree with you, but do you have any report to guide your speculations? Please i will be so delighted to understand this, and thanks 4 your contribution.


    Merged post follows:

    Consecutive posts merged

    I'm wondering why the low response. Pls share with me whatever your opinion, thanks all.


    Merged post follows:

    Consecutive posts merged

    I'm wondering why the low response. Pls share with me whatever your opinion, thanks all.

  2. In the most accepted hypothesis of oxidative phosphorylation, P . Mitchel gave an account on d transport of electrons in d electron transport chain on d inner membrane of d mitochondria , d generation of electrochemical gradient that drives d synthesis of ATP, the presence of d enzyme ATPase (a membrane bound protein in the inner membrane of the mitochondria) and hence the generation of ATP.

    In Rackers experiment, the membrane was distorted to isolate d fragments of d membrane from d protein which was d F1 Fo ATPase. On his conclusion, electron transport was possible on d fragments, witout phosphorylation, and there was phosphorylation by d protein fraction and then,d recontruction gives oxidative phosphorylation. My question is this:

    - Where was d energy derived from in Rackers experiment that caused d phosphorylation of ADP since d membrane is damaged? Cos an electrochemical gradient cannot be generated in d fragmented mitochondria.

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