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Is race a social construct? [ANSWERED: YES!]


Is race a social construct?  

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  1. 1. Is race a social construct?



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2 hours ago, Stevie Wonder said:
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The association between telomere length (TL) dynamics on cognitive performance over the life-course is not well understood. This study meta-analyses observational and causal associations between TL and six cognitive traits, with stratifications on APOE genotype, in a Mendelian Randomization (MR) framework. Twelve European cohorts (N=17 052; mean age=59.2±8.8 years) provided results for associations between qPCR-measured TL (T/S-ratio scale) and general cognitive function, mini-mental state exam (MMSE), processing speed by digit symbol substitution test (DSST), visuospatial functioning, memory and executive functioning (STROOP). In addition, a genetic risk score (GRS) for TL including seven known genetic variants for TL was calculated, and used in associations with cognitive traits as outcomes in all cohorts. Observational analyses showed that longer telomeres were associated with better scores on DSST (β=0.051 per s.d.-increase of TL; 95% confidence interval (CI): 0.024, 0.077; P=0.0002), and MMSE (β=0.025; 95% CI: 0.002, 0.047; P=0.03), and faster STROOP (β=-0.053; 95% CI: -0.087, -0.018; P=0.003). Effects for DSST were stronger in APOE ɛ4 non-carriers (β=0.081; 95% CI: 0.045, 0.117; P=1.0 × 10(-5)), whereas carriers performed better in STROOP (β=-0.074; 95% CI: -0.140, -0.009; P=0.03). Causal associations were found for STROOP only (β=-0.598 per s.d.-increase of TL; 95% CI: -1.125, -0.072; P=0.026), with a larger effect in ɛ4-carriers (β=-0.699; 95% CI: -1.330, -0.069; P=0.03). Two-sample replication analyses using CHARGE summary statistics showed causal effects between TL and general cognitive function and DSST, but not with STROOP. In conclusion, we suggest causal effects from longer TL on better cognitive performance, where APOE ɛ4-carriers might be at differential risk.

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Genome-wide association analysis advanced understanding of blood pressure (BP), a major risk factor for vascular conditions such as coronary heart disease and stroke. Accounting for smoking behavior may help identify BP loci and extend our knowledge of its genetic architecture. We performed genome-wide association meta-analyses of systolic and diastolic BP incorporating gene-smoking interactions in 610,091 individuals. 

2 minutes ago, Stevie Wonder said:

I already did. You ignored it.

Neither of the 2 above links about race being link to ancestry. You are providing research intended for other purposes is hopes of anecdotally inferring a point. Please, provided something in context to the discussing.

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18 minutes ago, Ten oz said:

Neither of the 2 above links about race being link to ancestry. You are providing research intended for other purposes is hopes of anecdotally inferring a point. Please, provided something in context to the discussing.

Race is defined by ancestry. Is always has been. Shakespeare used the word race to refer to lines of descent. Darwin explicitly defined the word race by shared genealogy. Anybody using an ancestry concept is using a race concept. It's just another word for the same thing.

You can say they don't use the word race and pretend they're using a different concept. But you'd be wrong. 

This is how scholars who use the word race define it.

http://en.rightpedia.info/w/Race#Definition_of_race

By ancestry.

You're just playing a silly semantic game. How do you define the word race? Some other way? Well then that's just you and you're not using words according to their common definition. Maybe you define race by "skin color" or something. Fine. Please bear in mind when I use the word race I mean shared ancestry. We're done here.

Edited by Stevie Wonder
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35 minutes ago, Stevie Wonder said:

We're done here.

!

Moderator Note

Agreed. And since you failed spectacularly to provide support for your arguments after repeated requests, you may not start another thread on this topic.

Thread closed.

 
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